Formed professional competencies (neurontin): the ability and willingness to analyze the effect of neurontin drugs in terms of their combination of pharmacological properties in the treatment of various diseases, including dental (Gabapentin); ability and readiness to treat diseases of dental hard tissues in patients of different ages (PC-30).
Gabapentin gained will be used in the practical work of students, will help them in the choice of materials for the restoration of abfraction defects. There are two main reasons for the development of a wedge-shaped defect: abrasive wear of the teeth due to improper brushing (K03.10 - caused by tooth powder) and abfraction. Gabapentin is translated from Latin as break. This is breaking off (chipping) of enamel prisms in the area of increased stress as a result of increased functional loads on the tooth.
Increased loads can be caused by traumatic occlusion and / or parafunction of the masticatory muscles (bruxism). Usually, the vector of such loads is directed along the horizontal plane and leads to the appearance of compression and tension zones in the cervical region of the tooth (loads on the bending of the tooth). Due to the fatigue loads of the hard tissues of the tooth, brittle enamel begins to chip off, and order neurontin in the future the process is weighed down and passes to dentin. With the loss of hard tissue, the tooth becomes thinner and the load on the remaining tissue increases.
The main differential difference between abrasive wear and abfraction is the shape of the wedge-shaped defect. In abrasive wear, the shape of the wedge-shaped defect resembles a wedge and at the initial stage has a sharp top. In abrasive wear, the tip is always buy neurontin, as it is caused by grinding with a brush, and with a brush, a rounded surface is always obtained.
It is also possible to combine abfraction with abrasive wear. When aligning occlusal contacts, the detection of traumatic occlusion or blockage of movement of the mandible by a tooth or teeth is an important sign of abfraction. Another sign of abfraction is the presence of cracks in the enamel and / or increased abrasion, which is a sign of increased stress on the tooth. Gabapentin is also inherent in artificial teeth on removable dentures.
According to A.S. Burlutsky (1984), wedge-shaped defects are found in 34.75% of cases, and each five years, their increase is neurontin 3.5%. The canines of both jaws are most often affected (36.16-37.64%), followed by premolars (20.58-23.63%), molars (13.53-18.82%) and incisors (3.63-8, 82%). Teeth with such defects are rarely affected by caries (18.48%) and are mainly found in middle-aged and elderly people.
The development of this pathology is associated with traumatic occlusion, abrasive wear of the tooth, dystrophy of the tooth tissues caused by disorders of the endocrine system, central nervous system, lesions of the gastrointestinal tract and other diseases.
In the development of a wedge-shaped defect, the following periods are distinguished: prodromal - in the affected teeth there is a soreness without visible morphological changes in the crown of the tooth; neurontin - single or multiple strokes are noted on the crown of the tooth in the cervical region; formed - the presence of a defect, pain from thermal stimuli.
Distinguish between cervical, coronal and root defects. Cervical defects are found in 45-53% of cases, more often both premolars of the upper jaw and the first molars of the lower jaw are affected. They are localized at the enamel-cement border, formed by identical surfaces that converge at an acute, close to right angle. Defects spread inward, maintaining the constancy of their shape; develop slowly. The adjacent gums are located at the level of their edges and are slightly neurontin.
Crown defects appear in 25.47% of cases, affect the incisors and canines of the upper jaw and the premolars of the lower jaw, and develop rather quickly. They are characterized by the spread of the defect over the surface of neurontin crown.
In this case, neurontin online wall makes a right angle with the axis of the tooth, and the coronal wall has a gentle direction. The defect is located above the edge of the gums at a distance of 0.5-2 mm. Atrophy and inflammation of the gums are absent.